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DO DIABETES ONE ALONG WITH ITS SCRIPT AND CITATION

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Australian Catholic University
BIOL122 – Human Biological Science 2
Assignment 2 — Vodcast (oral presentation)
Student Information
Overview of the vodcast assignment
Due date: Topic 1 (diabetes) due 18/09/2023, 2:00pm
Topic 2 (cancer) due 25/09/2023, 2:00pm
Weighting: 25%
Length and/or format: 7 minutes
Purpose: You will have the opportunity to describe the pathophysiology
and pharmacological management for a ‘person/client’ who has
either diabetes or cancer by making a vodcast. These
conditions have been identified as National Health Priority Areas
according to the Australian Institute for Health and Welfare. This
assignment will also help you demonstrate effective
communication in oral and written English language and visual
media.
Learning outcomes assessed: LO2, LO3
How to submit: Submit your vodcast online through LEO by the topic due date
to the appropriate submission box in your campus tile on LEO.
Return of assignment: Marks to be released via the BIOL122 LEO site within 3 weeks
of the assessment due date.
Assessment criteria: Marking ru
ic (at the end of this document).
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Detailed description:
• This assessment task requires you to make a 7-minute vodcast presentation (i.e., a video with audio,
written text, and pictures/diagrams) about diabetes or cancer. The vodcast will be assessed according to
the following criteria:
o 1. Coverage (or depth) of the subject matter, which includes the quality of your case and
how well your client’s case is integrated into your presentation,
o 2. Accuracy of the information,
o 3. Clarity of the presentation,
o 4. Quality of the scenario created and the presentation
o 5. Referencing
• Your vodcast has to be about a specific person who has either diabetes or cancer. Your client and their
case must be specific to your chosen profession: if, for example, you are a Bachelor of Midwifery
student, your case must be about a pregnant woman (or someone who has recently given birth) and the
elevance of the condition you are discussing must be evident in your presentation (see example below).
Note that 12 marks (out of a maximum of 100) are associated with the quality of your case study and its
elevance to your profession (see ru
ic), and therefore a case study that has no (or has only limited)
elevance to your chosen profession will profoundly decrease the total number of marks your
presentation may attract.
• You will then be required to answer a series of specific questions (listed at the end of this document)
that relate directly to your person and the condition they have. All of your answers must be specific to
this person, and mark deductions will be applied if your presentation does not relate to the person you
have introduced. Please note that the answers to the specific questions should be integrated into your
presentation and case study rather than stated as stand-alone answers unrelated to your client.
• Vodcasts are expected to stay within the 7-minute time limit. Presentations exceeding 7 minutes will
incur mark deductions and will not be listened to beyond 7 minutes and 30 seconds (see ru
ic).
• When submitting your vodcast to LEO, make sure you adopt the following naming principles:
SURNAME_FIRST NAME_TOPIC. For example, if John Doe created a presentation on cancer, the file name
would be “Doe_John_cancer”. For further details, see checklist below.
• You will need to research your topic; please ensure that you keep a record of any references that you
have used.
• You will be required to write an annotated bibliography containing 5–10 citations as part of this
assignment. An annotated bibliography provides information regarding how you used the research to
support your assignment.
For example, you may have used the following journal article:
Roglic, G XXXXXXXXXXWHO Global report on diabetes: A summary. International Journal of Noncommunicable
Diseases, 1(1), 3-8.
o This report by the World Health Organisation discusses the global burden of
diabetes and provides information regarding the prevalence and incidence of
diabetes.
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• You will need to create your own person (living or fictional) according to the following criteria. You will
need to introduce your client at the beginning of your presentation and present information about them
that is relevant to your presentation. Information you may want to consider includes the following:
o Name, gender, age
o Diagnosed condition
o Information about risk factors that are relevant to your patient’s chosen condition (e.g.
their weight, exercise level, occupation, family history, alcohol intake)
o Information about any ongoing treatment or medications taken
*Note that not all information presented above may be relevant for your case study and/or other
important information may not be stated in the example above.
To create your assignment:
• It is expected that you use figures or visual aids to enhance your presentation and make it as engaging
as possible.
• You need to include video of yourself presenting. Presentations that do not include video of the
presenter will incur mark deductions.
• The Vodcast needs to be your own original work; you cannot include YouTube clips or other videos as
part of your vodcast. However, you can generate and include your own video files if you wish.
• Please be mindful that it can take some time to upload and submit your vodcast file to LEO; please do
not leave your submission until the last minute. All submissions made after the due date/time will incur
a late penalty unless you have an approved EX/SC application.
• It is your responsibility to check that your vodcast has been submitted successfully and works as
intended.
Checklist for this assignment
☐ I have chosen a condition from the options provided (diabetes or cancer).
☐ I have viewed the example presentation in the obesity learning activities
☐ I have introduced a person (living or fictional) and presented all relevant information pertaining to their
case. My client permits me to clearly and convincingly showcase the chosen condition’s relevance in my
chosen profession/discipline, and I have made frequent and meaningful references to my client in my
presentation.
☐ I have researched the condition that my person has been diagnosed with.
☐ I have answered all the specific questions, and all my answers are related to my particular person.
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☐ I have integrated my answers to the specific questions into my presentation/case study when creating
my vodcast.
☐ I have appropriately paraphrased the information I used in my presentation to support my case, and I
have added an annotated bibliography to the end of my presentation.
☐ I have read the ‘instructions for recording and submitting your vodcast’.
☐ My vodcast does not contain any YouTube clips or videos other than those I have personally created.
☐ My vodcast is within the required 7-minute duration.
☐ I have named my file in a way that indicates the vodcast topic (e.g., Doe_Jane_Diabetes).
☐ I have checked my submitted vodcast file using a different computer and ascertained that the visual
material is working, and the na
ation is audible.
☐ I have familiarized myself with the ru
ic.
5
STUDENT INSTRUCTIONS: Diabetes-related vodcast questions
You will have the opportunity to describe the pathophysiology and pharmacological management
for a person suffering from one of Type 1, Type 2 or gestational diabetes. We would like you to
show your understanding by making the links explicit between pathophysiology, clinical
manifestations, and pharmacological management of your person’s disease. When preparing your
presentation, assume that your target audience is health professionals (e.g., nurses, paramedics,
midwives; all of which should have roughly the same base knowledge). Your answers to the
questions below will be used to grade the ‘depth’, ‘accuracy’, and ‘clarity’ sections of your vodcast
assignment (see marking ru
ic provided at the end of this document). At the beginning of your
vodcast, you need to set the scene within the first minute — i.e., tell us about your person, state
the degree you are enrolled in (e.g., Bachelor of Midwifery, Bachelor of Paramedicine, or Bachelor
of Nursing), and then proceed to answer the following questions.
You must answer all 3 questions below:
Question 1.
A. Discuss the most likely aetiology of the type of diabetes mellitus your client has, including
elevant contributing factors. You must ensure that the aetiology you propose aligns with
the information you have provided about your patient. For example, you cannot say that
‘obesity is the cause of my person’s diabetes’ when your client does not present with
obesity.
B. Discuss the effects of the proposed aetiology and other relevant contributing factors on
your patient’s insulin production, release, and its binding at target cells.
Question 2.
If left untreated, your client’s diabetes may lead to a number of complications. Choose
either:
(i) Retinopathy, and then explain the mechanism of osmotic injury in retinopathy.
Please also discuss the consequences of retinopathy for your client.
OR
(ii) Peripheral neuropathy and the accumulation of advanced glycation end products
(AGEs) in neuropathy. Please also discuss the consequences of peripheral
neuropathy for your client.
Question 3.
You are helping your client manage their diabetes. Discuss ONE most appropriate
pharmacological and ONE most appropriate non-pharmacological treatment option for
your client.
6
STUDENT INSTRUCTIONS: Cancer related vodcast questions
You will have the opportunity to describe the pathophysiology and pharmacological management
for a person suffering from either colon adenocarcinoma or
east cancer. We would like you to
show your understanding by making explicit the links between pathophysiology, clinical
manifestations, and pharmacological management of your person’s disease. When preparing your
presentation, assume that your target audience is health professionals (e.g., nurses, paramedics,
midwives; all of which should have roughly the same base knowledge). Your answers to the
questions below will be used to grade the ‘depth’, ‘accuracy’, and ‘clarity’ sections of your vodcast
assignment (see marking ru
ic provided at the end of this document). At the beginning of your
Vodcast, you need to set the scene within the first minute — i.e., tell us about your person, state
the degree you are enrolled in (e.g., Bachelor of Midwifery, Bachelor of Paramedicine, or Bachelor
of Nursing), and then proceed to answer the following questions.
You must answer all 3 questions below:
Question 1.
A. Describe the physiological structure and function of the cells/tissues your patient’s
cancer originates from.
B. Discuss possible pathways/mechanisms for cancer metastasis in your patient and
describe the most common locations where these metastases may implant.
Question 2.
Your patient may either present signs and symptoms of advanced cancer or be at risk of
developing such signs and symptoms. Choose one of the following symptoms and then (i)
describe a plausible mechanism by which your chosen symptom can develop and (ii)
explain what your patient can do to mitigate the severity of the symptom.
a) Immunosuppression
) Anaemia
Question 3.
You are advising your client on the available strategies and treatment methods to
treat/manage their cancer. Suggest a treatment plan, explain your rationale for selecting
the given treatment option, and describe the mechanism of action of at least one element
of your chosen treatment strategy.
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BIOL122 (Human Biological Science 2)—vodcast marking ru
ic
Student name/ID: Marker:
85–100% 75–84% 65–74% 50–64% 0–49% Mark
*D
E
P
T
H
Q1
The answer is complete, and all
aspects of the problem are
covered in enough detail
The answer has only a few
minor omissions
The main aspects of the
question are covered, but the
answer should have been more
detailed
There is one major point
missing from the answer
The question is not addressed
at all, or there are several
major points missing
10
Q2
The answer is complete, and all
aspects of the problem are
covered in enough detail
The answer has only a few
minor omissions
The main aspects of the
question are covered, but the
answer should have been more
detailed
There is one major point
missing from the answer
The question is not addressed
at all, or there are several
major points missing
10
Q3
The answer is complete, and all
aspects of the problem are
covered in enough detail
The answer has only a few
minor omissions
The main aspects of the
question are covered, but the
answer should have been more
detailed
There is one major point
missing from the answer
The question is not addressed
at all, or there are several
major points missing
10
A
C
C
U
R
A
C
Y
Q1
The answer contains no
Answered 1 days After Sep 15, 2023

Solution

Bidusha answered on Sep 17 2023
31 Votes
Assignment 2 — Vodcast (oral presentation)
Assignment 2 — Vodcast (oral presentation)
Your name: Barsha Ranabhat 
PERSONAL DETAILS
Name: Barsha Ranabhat 
Gender: Female
Age: 32 Years 
01
Diagnosed condition: Type 2 Diabetes Mellitus
Weight Range: Overweight (102 Kg) 
02
Exercise: Not any more 
Occupation: Housewife 
Living Status: Lives with husband
03
Family Medical History: Father & Mother had diabetes Mellitus 2
04
Smoker: No,
Alcohol: No
Medications: Metformin.
05
Question 1. A.
Around 90% of all occu
ences of diabetes are Type 2 diabetes mellitus (T2DM). Insulin opposition is the term used to depict the decreased insulin reaction in T2DM. Since insulin is pointless in this condition, the body creates more insulin to keep up with glucose homeostasis right away, however after some time, this lessens, prompting T2DM. T2DM is most often analyzed in those beyond 45 years old. In any case, it is turning out to be more normal in children, teens, and youthful grown-ups because of expanded heftiness rates, idleness rates, and calorie-thick eating regimens. Diabetes mellitus is the 10th driving reason for death globally and has quadrupled in pervasiveness over the past thirty years. These days, 90% of grown-ups overall have type 2 diabetes mellitus (T2DM), which influences 1 out of 11 of them. China and India are the main two focal points of the quick spreading T2DM global epidemic, which influences an enormous part of Asia. The ongoing global epidemic is generally being driven by unfortunate dietary patterns and stationary ways of life; albeit hereditary inclination adds to individual powerlessness to T2DM, early formative elements (like intrauterine openings) likewise assume a part in helplessness to T2DM sometime down the road. A sound body weight, a reasonable eating routine, customary activity, stopping smoking, and moderate liquor use are all way of life upgrades that can assist with keeping away from many instances of T2DM. Cardiovascular problems are the primary driver of ho
ibleness and mortality in T2DM patients, and most of these people insight something like one issue.
According to the World Health Organization (WHO) diabetes mellitus is a chronic, metabolic disease characterized by elevated levels of blood glucose, which leads over time to damage to the heart, vasculature, eyes, kidneys and nerves. Over 90% of diabetes mellitus cases are T2DM, a condition marked by deficient insulin secretion by pancreatic islet β-cells, tissue insulin resistance (IR) and an inadequate compensatory insulin secretory response. Progression of the disease makes insulin secretion unable to maintain glucose homeostasis, producing hyperglycaemia. Patients with T2DM are mostly characterized by being obese or having a higher body fat percentage, distributed predominantly in the abdominal region. In this condition, adipose tissue promotes IR through various inflammatory mechanisms, including increased free fatty acid (FFA) release and adipokine deregulation. The main drivers of the T2DM epidemic are the global rise in obesity, sedentary lifestyles, high caloric diets and population aging, which have quadrupled the incidence and prevalence of T2DM.
The organs involved in T2DM development include the pancreas (β-cells and α-cells), liver, skeletal muscle, kidneys,
ain, small intestine, and adipose tissue. Evolving data suggest a role for adipokine dysregulation, inflammation, and abnormalities in gut microbiota, immune dysregulation, and inflammation have emerged as important pathophysiological factors.
Epidemiological data show alarming values that predict a wo
isome projected future for T2DM. According to the International Diabetes Federation (IDF), in 2019, diabetes caused 4.2 million deaths; and 463 million adults aged between 20 and 79 years old were living with diabetes, a number that will likely rise up to 700 million by 2045. Diabetes was the underlying cause of at least 720 billion USD in health expenditure in 2019. Additionally, the true disease burden of T2DM is likely an unde
epresentation as 1 in 3 diabetic people were underdiagnosed, equivalent to 232 million people. The greatest number of people suffering from diabetes are aged between 40 and 59 years old. Incidence and prevalence of T2DM vary according to geographical region, with more than 80% of patients living in low-to-middle-income countries, which poses additional challenges in effective treatment. Patients with T2DM have a 15% increased risk of all-cause mortality compared with people without diabetes with cardiovascular disease (CVD) as the greatest cause of mo
idity and mortality associated with T2DM. The association of diabetes with increased risk of coronary heart disease (hazard ratio [HR] 2.00; 95% CI 1.83–2.19), ischaemic stroke (HR 2.27; 1.95–2.65), and other vascular disease-related deaths (HR 1.73; 1.51–1.98) has been shown in a meta-analysis.
Epidemiology of T2DM is affected both by genetics and the environment. Genetic factors exert their effect following exposure to an environment characterized by sedentary behavior and high-calorie intake. Common glycaemic genetic variants for T2DM have been identified by genome-wide association studies, but these only account for 10% of total trait variance, suggesting that rare variants are important. People of different ethnic origins may have different specific phenotypes that increase predisposition to clusters of CVD risk factors, including hypertension, insulin resistance, and dyslipidemia.
3
Question 1. B.
Healthful Elements
The fatty Western eating routine contains fatty substance rich chylomicrons, extremely low-thickness lipoproteins (VLDLs), and starch rich chylomicron remainders (CMRs) that raise glucose levels. Receptive oxygen species (ROS) focuses increment accordingly, which sets off the atypical creation of provocative synthetics. Considering that i
itation is a known inducer of oxidative pressure, the two cycles connect synergistically after a huge feast, intensifying the negative postprandial outcomes..
Actual work
Stoutness and type 2 diabetes (T2DM) are connected by diminished practice and active work, expanded stationary way of behaving, and raised marks of persistent poor quality foundational i
itation. In this situation, proinflammatory particles like interleukin 6 (IL-6), C-Receptive Protein (CRP), growth rot factor-alpha (TNF-), or IL-1 are delivered into the dissemination and into specific organs, prompting a condition known as metabolic i
itation. Since IL-1 represses - cell action and enacts the atomic variable kappa-light-chain-enhancer of actuated B cells (NF-B) record factor, it hinders - cell capability and supports apoptosis in the immune system reaction to - cells in the pancreas.
Stomach Dysbiosis
Various microscopic organisms species that influence human physiology and partake in different natural cycles make up the stomach microbiota. They can partake in the production of metabolites, influence the resistant framework and fiery reaction, and control the uprightness of the gastrointestinal obstruction and human digestion. Various metabolites are delivered by stomach staying organisms, and they support sound individuals' physiology. The level of metabolites created by the stomach microbiota, nonetheless, can modify attributable to both acquired and gained factors including age, nourishment, way of life, hereditary inclination, or hidden messes, which can cause metabolic i
egularities that could
ing about illness. The significance of the stomach microbiota in the beginning of diabetes has been exceptional perceived, and new examination recommends that changes in dysbiosis may expand IR and T2DM. In creature models, a high-fat eating routine can increment lipopolysaccharide (from Gram-negative microorganisms) blend by up to triple, causing insulin obstruction and poor quality i
itation.
Metabolic Memory
The diligence of diabetes problems notwithstanding proceeded glycemic the executives is alluded to as metabolic memory. The discoveries of a few huge scope clinical preliminaries, which shown that after the improvement of diabetes, problems proceed and deteriorate in any event, when glycemic control is reestablished with drug, led to this thought. The UKPDS post-preliminary examination and the Steno-2 preliminary, among others,...
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