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case study scenerio - final.pdf NURBN 2012: Nursing Practice 3 - Pathophysiology and Pharmacology Applied to Nursing Assessment Task 2 – Clinical Scenario Assignment Due Date: Friday 8th May at 5pm...

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case study scenerio - final.pdf

NURBN 2012: Nursing Practice 3 - Pathophysiology and Pharmacology Applied to Nursing
Assessment Task 2 – Clinical Scenario Assignment

Due Date: Friday 8th May at 5pm
Word Count: 2000 words +/- 10%
Format: Report style, with introduction and conclusion. A cover sheet is not required. You will be
equired to submit this assignment through Turnitin.
Directions
For this assessment task, you are required to write a 2000 word report answering the questions
from the scenario. You will need to explore the pathophysiology, pharmacology and psychosocial
aspects of the scenario and demonstrate your understanding in the answers you provide. Your
answers should be informed by your reading of cu
ent research and literature.

A report format includes an introduction and conclusion, but headings are used in the body of the
text. Use the question you are answering as your heading. Do not use dot points for your answers.
Do not write in the first person. Appropriately referenced and labelled tables, diagrams or images
may be used in the body of the paper. You may reproduce the tables used in the questions in your
answers.

Use APA referencing (6th) throughout your assignment. References must be cu
ent, preferably from
the past five years. You are required to cite no less than 10 references, and the majority of these
should be cu
ent journal articles. We are looking at information informing cu
ent clinical practice,
and your choice of references must reflect this.

Consider the quality of the references you use. Wikipedia, Web MD, the Better Health Channel and
logging websites are not acceptable references. Any non-reliable sources in your reference list will
not count toward the number of required references, and this will result in a loss of marks. At this
point in your BN you are expected to engage with best practice literature.

Criteria for grading

Marking of this assessment task will be undertaken by academic and teaching staff. Pre and post
moderation will be undertaken for this assessment task.

The marking ru
ic for the assignment is provided on Moodle. Marks will be allocated for each
section according to the ru
ic. Use the ru
ic as a guide when writing your assignment to identify
the depth of the answer expected.
Case Scenario
Russell is a truck driver aged 68 years whos admitted to Monash Health with
eathlessness.

History of presenting complaints

He describes progressive dyspnoea that he has had for the past three months and is now
eathless
when showering and dressing. For the past week, he has felt more comfortable overnight sleeping
on three pillows. He denies any chest pain.

You have taken Russell’s history and vitals and made a Med call to review Russell.


Past medical history

Russell says he had a heart attack 15 years ago, which was treated with a stent, and has had no chest
pain since then.

Russell has Chronic Obstructive Pulmonary Disease (COPD) for the past 30 years.

He also has had problems with high blood pressure, diabetes and cholesterol.

Medication history

His medications are as following -

 Aspirin 100mg oral daily,

 Salbutamol 2 – 4 puffs PRN

 Budesonide/Efomoterol fumarate dehydrate 2 puffs daily

 Perindopril 5 mg in the morning daily

 Atenolol 50 mg daily in the morning

 Metformin 500mg oral BD

 Atorvastatin 40 mg daily.


Russell is ma
ied and has two children. He used to smoke 20 cigarettes per day since he was 18
years old, but stopped smoking when he had his heart attack. He drinks a small amount of alcohol.

Observation/On Examination

On examination his
 Heart rate (HR) is 90 bpm and regular
 Blood pressure (BP) is 150/90 mmHg
 Jugular venous pressure (JVP) is slightly elevated
 He has mild oedema in his both legs
 BMI 26 m2/kg
 Skin – sweaty and pale
 RR 26, regular
 SpO2 94% RA
 Temp. 37o C
 BSL 5.5

Duty doctor examined him and confirmed Russell develop heart failure. He orders some laboratory
investigations, CXR and Echocardiogram, 2L oxygen, Tab Frusemide 40 mg in the morning and Tab
Spironolactone 25 mg orally once a day.

Biochemistry results are:

 Na 135 mmol/L [134 to 145 mmol/L]
 K 4.2 mmol/L [3.5 to 5.0 mmol/L]
 Urea 8.9 mmol/L; [2.5 to 7.1 mmol/L]
 Creatinine 98 μmol/L [53 to 106 μmol/L]
 Total cholesterol (TC) 6.8 mmol/l, [<5.5 mmol/L]
 Low density lipoprotein (LDL) 5.0 mmol/L [2.0 mmol/L]
 High density lipoprotein (HDL) 1.0 mmol/L [> 1.0mmol/L]
 Triglycerides (TG) 2.1 mmol/L [< 2.0 mmol/L]

Full blood count is normal

Chest X-ray
A chest X-ray shows an increased cardiothoracic ratio (dilated heart) and obliteration (not visible) of
cardiophrenic and costophrenic angles in the lung fields suggestive of pulmonary oedema and heart
failure

Echocardiogram shows a dilated left ventricle with severe systolic dysfunction (left ventricular
ejection fraction (LVEF) 25%.

Russell’s dyspnoea improves with Frusemide and Spironolactone


Case scenario questions

Q1. Discuss risk factors that contributed to Russell developing heart failure (10 marks)

Q2. Describe pathophysiology of right and left sided heart failure using clinical presentation and
examination findings of Russell (20 marks)

Russell has long history of COPD.

Q3. Explain the term ‘acute exace
ation of COPD’ [8 marks]
What factors put patients like Russell at high risk for exace
ations of COPD? [8 marks]

Q4. Use the table below (or something similar) to discuss the following drugs:

Perindopril, Spironolactone, Budesonide/Fomoterol fumarate dehydrate puffs. Do not list
gastrointestinal upsets (such as nausea and vomiting) as complications/side effects or nursing
considerations. Only include information that can be directly applied to Russell (24 marks for
medications)


Generic name

Perindopril Spironolactone Budesonide/Fomoterol
fumarate dehydrate
puffs
Drug group (1 mark)
Mechanism of action
(3 marks)
Complications/side
effects (2 major) (1
mark each)
Nursing
considerations (2
major) (1 mark each)

Q5. What non-pharmacological recommendations, if any, do you make for Russell? (20 marks – 5
marks for each)

a. For heart failure

. To prevent exace
ation of COPD

c. To prevent pneumonia

d. To reduce his high cholesterol level

Presentation, readability and ref – [10 marks]
NURBN 2012 Clinical Scenario Assignment Marking Ru
ic.pdf
NURBN 2012 Clinical Scenario Assignment Marking Ru
ic /2020
Criteria High Distinction 80 –100% Distinction 70 – 79% Credit 60 – 69% Pass 50 – 59% Fail 0 – 49%
Discussion of risk factors that
contributed to Russell
developing heart failure [10
marks]
Co
ect identification of all
isk factors and explained
them with evidence.
Risk factors not mostly
co
ectly identified and
some evidence provided
Risk factors partially identified
and explained partially with
evidence
Risk factors inco
ect and
cursory use of evidence
Risk factors inco
ect OR
question not attempted
Describe pathophysiology of
ight and left sided heart failure
using clinical presentation and
examination findings of Russell
[20 marks]
Excellent explanation of
pathophysiology of right
and left sided heart failure.

Excellent linking of
Russell’s presentation to
his underlying pathology
Very good explanation
pathophysiology right and
left sided heart failure.

Excellent linking of Russell’s
presentation to his
underlying pathology

Good explanation
pathophysiology right and left
sided heart failure.

Good linking of Russell’s
presentation to his underlying
pathology

Inadequate explanation
pathophysiology right and left
sided heart failure.

Inadequate excellent linking
of Russell’s presentation to
his underlying pathology

Pathophysiology question
not attempted or
explanation mostly
inco
ect
Explain the term ‘acute
exace
ation of COPD’ [8
marks]
Explain factors that can likely to
put Russell at high risk for
exace
ations of COPD [8
marks]
Excellent explanation of
“acute exace
ation of
COPD”

Excellent discussion of risk
factors that can likely to
put Russell at high risk for
exace
ations of COPD.
Very good explanation of
“acute exace
ation of
COPD”

Very good discussion of risk
factors that can likely to put
Russell at high risk for
exace
ations of COPD.

Good explanation of “acute
exace
ation of COPD”

Good discussion of risk factors
that can likely to put Russell at
high risk for exace
ations of
COPD.
Inadequate explanation of
“acute exace
ation of COPD”

Inadequate explanation of risk
factors that can likely to put
Russell at high risk for
exace
ations of COPD.
No attempt to answer the
clinical scenario
Identification of
pharmacological issues relevant
to clinical scenario – three
drugs worth 8 marks each [24
marks]
All relevant
pharmacological concepts
and mechanisms of action
are included and co
ect.
All relevant
contraindications, adverse
eactions, nursing
considerations and patient
education points included

All relevant pharmacological
concepts and mechanisms
of action are included and
co
ect. Most relevant
contraindications, adverse
eactions, nursing
considerations and patient
education points included

Most relevant pharmacological
concepts and mechanisms of
action are included and
co
ect. Some relevant
contraindications, adverse
eactions, nursing
considerations and patient
education points included

Few relevant pharmacological
concepts and mechanisms of
action are included or co
ect.
Answered Same Day May 07, 2021

Solution

Sunabh answered on May 12 2021
163 Votes
Running Head: NURSING        1
NURSING        13
NURBN 2012: NURSING PRACTICE 3 - PATHOPHYSIOLOGY AND PHARMACOLOGY APPLIED TO NURSING
ASSESSMENT TASK 2 – CLINICAL SCENARIO ASSIGNMENT
Table of Contents
Introduction    3
Risk Factors That Contributed To Russell Developing Heart Fail    3
Pathophysiology of Right and Left Sided Heart Failure    4
Acute Exace
ation of COPD and Factors that Put Patients like Russell at High Risk for the Disease    5
Identification of Pharmacological Issues and Relevant Drugs    6
Non-Pharmacological Recommendations for Russell Related To    9
Conclusion    10
References    12
Introduction
Russell, a 68-year-old truck driver, was admitted Monash Health due to
eathlessness. He has been reported to suffer from Chronic Obstructive Pulmonary Disease (COPD) for the past 30 years and cu
ently Russell described progressive dyspnoea from past 3 months and he gets
eathless even during showering or dressing. Russell had a heart attack 15 years ago along with issues such as, diabetes, blood pressure and cholesterol. This report will focus upon the pathophysiology and pharmacological aspects required during treatment.
Risk Factors That Contributed To Russell Developing Heart Fail
It would be essential to consider that Russell’s chest X-ray reflected dilated heart and obliteration of costophrenic and cardiophrenic angles, which were the suggestive of heart failure. COPD is chronic lung disease and instead, it is a group of diseases, which blocks the airway as a result, patient suffers from
eathlessness and shortness of
eath. Russell has been suffering from COPD for more than 30 years and as suggested by de Miguel Diez, Morgan and Garcia, (2013), COPD has been commonly associated with heart failure. Both these conditions might coexist with in the clinical practice and heart failure has been found to be prevalent in around 20% of COPD cases.
Further, it would also be essential to consider that both the diseases has some common risk factors such as, cigarette smoking, systemic inflammation along with advanced age (Pirina et al., 2017). Russell has reported that, he used to smoke 20 cigarettes every day since, he was 18 year old; however, he stopped after he had a heart attack 15 years ago. This reflects that, despite of suffering from COPD, Russell was smoking for 15 years and this could be considered as one of the major factors behind heart failure. Likewise, Russell’s heart attack was treated with a stent; in order to open up the blockages and allow blood flow. Consumption of alcohol could also be considered as a risk factor for the development of heart failure and Russell has been reported to consume small amount of alcohol.
Pathophysiology of Right and Left Sided Heart Failure
    Congestive heart failure (CHF) is a condition when the heart too weak in order to pump blood out to rest of the body or organs. Heart supplied oxygenated blood to the body organs while it pumps deoxygenated blood back to lungs. Therefore, this might result into fluid build-up and blood might pump back to heart and lungs, causing shortness of
eath. All of these symptoms have been reported in Russell. Likewise, it would also be essential to consider that CHF patients do not face difficulty in
eathing when they are at rest rather, they might face shortness of
eath when they perform certain function.
    It would be essential to consider that patients suffering from COPD might have damaged airways and i
itation in the sacs or lungs where ca
on dioxide and oxygen are exchanged. Bosch et al. (2017) mentioned that, when a patient suffering from COPD exhales, this damage the airways does not allow oxygen to be fully released before the next
eath might be taken and this results into shortness of
eath. However, COPD and heart failure are two distinct concerns associated with two different organs that are, heart and lungs they could present similar symptoms. There are two different forms of heart failure associated with COPD and these include left sided heart failure and right sided heart failure.
Left-Sided Heart Failure –
It would be essential to consider that there is no direct connection between left sided heart failure and COPD still; these two conditions might exist together. This is majorly because, as presented above also that low oxygen levels in blood due to COPD places additional stress on heart. This condition might worsen the symptoms of left sided heart failure and could further result into fluid build-up in lungs, ultimately aggravating COPD symptoms.
Right-Sided Heart Failure –
Development of right-sided heart failure occurs in severe cases. Severe cases of COPD such as, in case of Russell where COPD pertains for more than 30 years could lead to the development of right-sided heart failure. This occurs due to the drop in oxygen levels...
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