Great Deal! Get Instant $10 FREE in Account on First Order + 10% Cashback on Every Order Order Now

2808NRS Assessment 2: Trimester 2, 2018 Weighting: 30% Due Date: Week 6; Monday 20 th August by 5pm Learning Outcomes Assessed: 1, 2, 3 & 4 Task aim/purpose: The aim of this case-based assignment is...

1 answer below »
2808NRS Assessment 2: Trimester 2, 2018
Weighting: 30%
Due Date: Week 6; Monday 20
th
August by 5pm
Learning Outcomes Assessed: 1, 2, 3 & 4
Task aim/purpose:
The aim of this case-based assignment is to allow you to demonstrate your clinical reasoning skills, your capacity to
differentiate normal from abnormal presentation and to identify appropriate (evidence-based) investigations and
treatment modalities associated with the assigned case-study, which are skills that are crucial to safe and effective
nursing practice.
Individual, Written Assignment, concept map
Total Marks: 100
Task description:
 For this task you need to create a single (1) slide (using PowerPoint) colour coded concept map and provide a
750 word explanation, demonstrating analysis of the case study scenario emailed to you in week 1. A
PowerPoint template file will be made available for you to customise based on your assigned case scenario.
 In your concept map you must include:
1) Your Interpretation of the patient’s risk factors (from the case-study scenario) and determine how these
isk factors relate to the diagnosed disease/disorder using evidence based literature;
2) An outline of the links between the aetiology, cellular pathology and the pathophysiology of the diagnosed
disease;
3) A description of how the pathophysiology of the disease/disorder accounts for the patient’s clinical
manifestations (described in the case-study scenario); and
4) An analysis and interpretation including evidence-based research to suggest appropriate diagnostic
assessments and treatment modalities for the patient’s diagnosis.
 In your 750 word written explanation you must:
1) Explain the links between the patient’s risk factors and aetiology to account for the disease’s/disorder’s
pathophysiology.
2) Describe how the disease’s/disorder’s pathophysiology manifests through the patient’s signs and
symptoms (clinical manifestations).
3) Justify your suggested diagnostic tests and treatment modalities listed in the concept map on their
elevance and appropriateness for the diagnosed disease/disorder.
Due Date:
Submission via Turnitin within the course [email protected] site by Monday week 6, 20th August 2018, 5pm.
Other elements:
 The concept map (colour coded) should be a maximum of 1 PowerPoint slide in length and the 750 words
written explanation (excluding reference list) should follow on the next slide/s;
 The submitted file MUST BE a PowerPoint file
 You will be presented with a case study scenario electronically (via email) by Friday 20th July (week 1);
 Use APA 6th edition for in-text referencing and in the reference list;
 Reference list should be on separate slide/s following the 750 word written explanation;
 Ensure that the majority of your references have been published within the last 10 years;
 Marking, moderation, and student feedback will be provided electronically based on a marking criteria that is
in accordance with University assessment policy;
 5% late penalties apply for each working day after submission date/time; if you submit your assignment more
than five days after the due date, you will receive zero.
 Please ensure you receive a turnitin receipt (take a screen shot) after submitting your assignment and please
check you have uploaded the co
ect assessment piece.
 Always refer to the marking criteria as this will assist you to determine the depth of response for each section;
 No coversheet is required; instead complete the eAssignment Cover Sheet Quiz before submitting your
assignment.
Refer to the Health Writing and Referencing guide for more information:
Griffith Health Writing & Referencing Guide.
https:
learn.griffith.edu.au/webapps
lackboard/content/listContentEditable.jsp?content_id=_1760774_1&course_id=_47507_1#contextMenu
https:
learn.griffith.edu.au/webapps
lackboard/content/listContentEditable.jsp?content_id=_1760774_1&course_id=_47507_1#contextMenu
http:
www.apastyle.org/learn/tutorials
asics-tutorial.aspx
https:
sites.google.com/a/griffith.edu.au/griffith-health-writing-and-referencing-guide/Assignment-Presentation-Formatting-Guidelines
https:
sites.google.com/a/griffith.edu.au/griffith-health-writing-and-referencing-guide/Assignment-Presentation-Formatting-Guidelines
2804NRS: Individual, Written Assignment, Concept Map Marking Criteria
Content Possible
mark
Mark
achieved
Brief comments
Presentation, written communication
 Concept map and written explanation has a logical
structure.
 Uses academic language throughout.
 No spelling/grammar mistakes
 Reference list included on separate page/s
 Concept map is appropriately colour coded with a
espective key.
 Co
ect word count (1 PowerPoint slide for concept
map & no more than 750 words for explanation).
 PowerPoint file submitted (not word, JPEG, PDF
etc.)
10
Concept map
 Clearly identifies ALL the risk factors from the
scenario relating to the patient’s diagnosis.
 Co
ectly links these risk factors and the aetiology
with the disease’s/disorder’s pathophysiology.
 Clearly identifies the pertinent disease’s/disorder’s
pathophysiological steps and logically sequences
them.
 Clearly identifies ALL the clinical manifestations
from the scenario and links them accurately with
the disease’s/disorder’s pathophysiology.
 Identifies and links evidence-based diagnostic
assessments and treatment modalities within the
oader concept map of the patient’s diagnosis.
35
Written explanation
 Clearly explains how the listed risk factors and
aetiology contributes to the patient’s diagnosis.
 Provides a detailed analysis of the
pathophysiological justification for each of the
patient’s clinical manifestations.
 The listed diagnostic assessments and treatment
modalities are justified for their clinical
appropriateness relating to the patient’s diagnosis,
and are substantiated with appropriate citations.
45

Use of references
 Written explanation includes reputable, cu
ent and
appropriate references.
 No fewer than 10 references used.
 Referencing as per APA Style 6th Edition, in-text
and in reference list.
 All in-text citations are listed in the reference list
10
Total Mark 100
Weighting for assignment 30%
Answered Same Day Jul 20, 2020 2804NRS

Solution

Sumayya K. answered on Jul 28 2020
105 Votes
Slide 1
HF
SMOKING
AGE
SYSTEMIC INFLAMMATION
COPD induced PH
VASCULAR REMODELLING
ENDOTHELIAL DYSFUNCTION
INFLAMMATION
SKELETAL MUSCLE ATROPHY
DYSPNEA
CHEST PAIN
JVD
PARASTERNAL HEAVE
HEPATOMEGALY
PROMINENT S2
OVERLAP SYNDROME
NATRIURETIC PEPTIDES
RHC
MRI
PFT
O2 THERAPY
Β- BLOCKERS & β-AGONISTS
DIURETICS
VASODILATORS
ACE & ARB
IVABRADINE
Pathophysiology
Risk Factors
Aetiology
Clinical manifestations
Diagnostic tests
Treatment
CONCEPT
MAP
OF
COPD related
HF
KEYS
COPD
HYPOXIA
VASO
CONSTRICTION
PULMONARY ARTERY
CHANGES
RV OVERLOAD
TNFa
ACTIVATE CRP
ATHERO
SCLEROSIS
INTRODUCTION
Heart Failure (HF) is often known to coexist with chronic obstructive pulmonary disease in the clinical scenario with more
than 20% of HF patients affected by COPD.(De Miguel, Morgan, and García, 2013). The common risk factors and
pathogenic mechanisms contribute to the complication causing diagnostic and treatment difficulties
(Hawkins, Virani, and Ceconi, 2013).
RISK FACTORS
Both HF and COPD share common risk factors like smoking, age and systemic inflammation (De Miguel, Morgan, and
García, 2013), (Güder et. al., 2014).
ETIOLOGY
Pulmonary hypertension is a major cause of concu
ent HF and COPD (I
ahim, 2016).
Endothelial dysfunction, vascular remodeling, skeletal muscle atrophy and inflammatory reactions also contribute to the
coexistence. (Soliman et. al., 2015), (Celutkiene et. al., 2016)
PATHOPHYSIOLOGY
The hypoxia due to COPD causes constriction in the pulmonary arteries. This leads to structural changes across all layers
of the pulmonary arterial walls which, in effect, impact right ventricular function causing heart failure. (MacNee, 2010)
Evidence suggests that stable COPD is linked to low-grade systemic inflammation with elevated levels of circulating
cytokines. TNFa trigger cardiac and pulmonary inflammation by activating secondary mediators like C-reactive protein (CRP)
and pro-inflammatory cytokines causing coronary atherosclerosis which in turn leads to heart failure. Also, functional genes
capable of producing both COPD and cardiovascular conditions have not been identified yet. (Hawkins, Virani, and Ceconi,
2013)
Studies have also shown that sleep apnoea along with COPD, known as overlap syndrome attribute to vascular endothelial
dysfunction, high inflammatory mediators and atherosclerosis. (De Miguel, Morgan, and García, 2013)
DIAGNOSIS
Of the COPD...
SOLUTION.PDF

Answer To This Question Is Available To Download

Related Questions & Answers

More Questions »

Submit New Assignment

Copy and Paste Your Assignment Here